11th International Congress of Human Genetics

Session Details

Gene Action - Mouse Models

Thursday, 10 August 2006 11:00 -13:00

P3/P4/P5

Co-Chair David L Nelson Baylor College of MedicineUnited States
Co-Chair Dr Emma Whitelaw Queensland Institute of Medical Research, BrisbaneAustralia

Increased dosage of DSCR1 and DYRK1A destabilizes NFAT regulation and accounts for Down syndrome phenotypes
  • Uta Francke, Stanford University, United States
  • Overdose of a short HSA21 segment causes reduction of REST transcript levels in humans and mouse Down syndrome models
  • Prof Dean Nizetic, Barts & The London, Queen Mary College Medical School, United Kingdom
  • The fragile X gene FMR1 participates in control of the mammalian circadian clock
  • David L Nelson, Baylor College of Medicine, United States
  • An inducible mouse model of RNA toxicity in myotonic muscular dystrophy
  • Mani S Mahadevan, University of Virginia, United States
  • An ENU screen in the mouse reveals the role of epigenetics in disease phenotypes
  • Ms Alyson Ashe, University of Sydney, Australia
  • Disruption of Bardet-Biedl syndrome ciliary proteins perturbs planar cell polarity in vertebrates
  • Dr Alison J Ross, Institute of Child Health, University College London, United Kingdom
  • Characterization of a hypomorphic allele of the FKBP8 gene
  • Dr Richard H Finnell, The Texas Institute for Genomic Medicine, United States
  • A gene for speed: The ACTN3 R577X polymorphism influences muscle performance
  • Daniel G MacArthur, Institute for Neuromuscular Research, Children's Hospital at Westmead, Sydney, Australia, Australia
  • Published on Tuesday, 1 August 2006 by the Professional Conference Organiser